W. Sun Et Al. , "TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis via increased aerobic glycolysis and HIF1-alpha stabilization," CANCER RESEARCH , vol.70, 2010
Sun, W. Et Al. 2010. TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis via increased aerobic glycolysis and HIF1-alpha stabilization. CANCER RESEARCH , vol.70 .
Sun, W., LIU, Y., GLAZER, C. A., SHAO, C., BHAN, S., Demokan, S., ... ZHAO, M.(2010). TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis via increased aerobic glycolysis and HIF1-alpha stabilization. CANCER RESEARCH , vol.70.
Sun, Wenyue Et Al. "TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis via increased aerobic glycolysis and HIF1-alpha stabilization," CANCER RESEARCH , vol.70, 2010
Sun, Wenyue Et Al. "TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis via increased aerobic glycolysis and HIF1-alpha stabilization." CANCER RESEARCH , vol.70, 2010
Sun, W. Et Al. (2010) . "TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis via increased aerobic glycolysis and HIF1-alpha stabilization." CANCER RESEARCH , vol.70.
@article{article, author={Wenyue Sun Et Al. }, title={TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis via increased aerobic glycolysis and HIF1-alpha stabilization}, journal={CANCER RESEARCH}, year=2010}