W. Sun Et Al. , "TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis through increased aerobic glycolysis and HIF1α stabilization," Clinical Cancer Research , vol.16, no.3, pp.857-866, 2010
Sun, W. Et Al. 2010. TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis through increased aerobic glycolysis and HIF1α stabilization. Clinical Cancer Research , vol.16, no.3 , 857-866.
Sun, W., LIU, Y., GLAZER, C. A., SHAO, C., BHAN, S., Demokan, S., ... ZHAO, M.(2010). TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis through increased aerobic glycolysis and HIF1α stabilization. Clinical Cancer Research , vol.16, no.3, 857-866.
Sun, Wenyue Et Al. "TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis through increased aerobic glycolysis and HIF1α stabilization," Clinical Cancer Research , vol.16, no.3, 857-866, 2010
Sun, Wenyue Et Al. "TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis through increased aerobic glycolysis and HIF1α stabilization." Clinical Cancer Research , vol.16, no.3, pp.857-866, 2010
Sun, W. Et Al. (2010) . "TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis through increased aerobic glycolysis and HIF1α stabilization." Clinical Cancer Research , vol.16, no.3, pp.857-866.
@article{article, author={Wenyue Sun Et Al. }, title={TKTL1 is activated by promoter hypomethylation and contributes to head and neck squamous cell carcinoma carcinogenesis through increased aerobic glycolysis and HIF1α stabilization}, journal={Clinical Cancer Research}, year=2010, pages={857-866} }