K. Freeman Et Al. , "Conditional activation of fibroblast growth factor receptor (FGFR) 1, but not FGFR2, in prostate cancer cells leads to increased osteopontin induction, extracellular signal-regulated kinase activation, and in vivo proliferation," CANCER RESEARCH , vol.63, no.19, pp.6237-6243, 2003
Freeman, K. Et Al. 2003. Conditional activation of fibroblast growth factor receptor (FGFR) 1, but not FGFR2, in prostate cancer cells leads to increased osteopontin induction, extracellular signal-regulated kinase activation, and in vivo proliferation. CANCER RESEARCH , vol.63, no.19 , 6237-6243.
Freeman, K., Gangula, R., Welm, B., Ozen, M., Foster, B., Rosen, J., ... Ittmann, M.(2003). Conditional activation of fibroblast growth factor receptor (FGFR) 1, but not FGFR2, in prostate cancer cells leads to increased osteopontin induction, extracellular signal-regulated kinase activation, and in vivo proliferation. CANCER RESEARCH , vol.63, no.19, 6237-6243.
Freeman, KW Et Al. "Conditional activation of fibroblast growth factor receptor (FGFR) 1, but not FGFR2, in prostate cancer cells leads to increased osteopontin induction, extracellular signal-regulated kinase activation, and in vivo proliferation," CANCER RESEARCH , vol.63, no.19, 6237-6243, 2003
Freeman, KW Et Al. "Conditional activation of fibroblast growth factor receptor (FGFR) 1, but not FGFR2, in prostate cancer cells leads to increased osteopontin induction, extracellular signal-regulated kinase activation, and in vivo proliferation." CANCER RESEARCH , vol.63, no.19, pp.6237-6243, 2003
Freeman, K. Et Al. (2003) . "Conditional activation of fibroblast growth factor receptor (FGFR) 1, but not FGFR2, in prostate cancer cells leads to increased osteopontin induction, extracellular signal-regulated kinase activation, and in vivo proliferation." CANCER RESEARCH , vol.63, no.19, pp.6237-6243.
@article{article, author={KW Freeman Et Al. }, title={Conditional activation of fibroblast growth factor receptor (FGFR) 1, but not FGFR2, in prostate cancer cells leads to increased osteopontin induction, extracellular signal-regulated kinase activation, and in vivo proliferation}, journal={CANCER RESEARCH}, year=2003, pages={6237-6243} }