S. Oktayoğlu Et Al. , "Glucotoxicity Causes Pancreatic Beta and Duct Cells to Mutually Suppress Each Other's Function via miR -335 Targeted Runx2 and Insulin-Mediated Mechanism," İstinye Üniversitesi Kök Hücre ve Doku Mühendisliği Sempozyumu , İstanbul, Turkey, pp.86-87, 2021
Oktayoğlu, S. Et Al. 2021. Glucotoxicity Causes Pancreatic Beta and Duct Cells to Mutually Suppress Each Other's Function via miR -335 Targeted Runx2 and Insulin-Mediated Mechanism. İstinye Üniversitesi Kök Hücre ve Doku Mühendisliği Sempozyumu , (İstanbul, Turkey), 86-87.
Oktayoğlu, S., Sancar, S., Karatuğ Kaçar, A., & Bolkent, Ş., (2021). Glucotoxicity Causes Pancreatic Beta and Duct Cells to Mutually Suppress Each Other's Function via miR -335 Targeted Runx2 and Insulin-Mediated Mechanism . İstinye Üniversitesi Kök Hücre ve Doku Mühendisliği Sempozyumu (pp.86-87). İstanbul, Turkey
Oktayoğlu, Selda Et Al. "Glucotoxicity Causes Pancreatic Beta and Duct Cells to Mutually Suppress Each Other's Function via miR -335 Targeted Runx2 and Insulin-Mediated Mechanism," İstinye Üniversitesi Kök Hücre ve Doku Mühendisliği Sempozyumu, İstanbul, Turkey, 2021
Oktayoğlu, Selda Et Al. "Glucotoxicity Causes Pancreatic Beta and Duct Cells to Mutually Suppress Each Other's Function via miR -335 Targeted Runx2 and Insulin-Mediated Mechanism." İstinye Üniversitesi Kök Hücre ve Doku Mühendisliği Sempozyumu , İstanbul, Turkey, pp.86-87, 2021
Oktayoğlu, S. Et Al. (2021) . "Glucotoxicity Causes Pancreatic Beta and Duct Cells to Mutually Suppress Each Other's Function via miR -335 Targeted Runx2 and Insulin-Mediated Mechanism." İstinye Üniversitesi Kök Hücre ve Doku Mühendisliği Sempozyumu , İstanbul, Turkey, pp.86-87.
@conferencepaper{conferencepaper, author={Selda OKTAYOĞLU Et Al. }, title={Glucotoxicity Causes Pancreatic Beta and Duct Cells to Mutually Suppress Each Other's Function via miR -335 Targeted Runx2 and Insulin-Mediated Mechanism}, congress name={İstinye Üniversitesi Kök Hücre ve Doku Mühendisliği Sempozyumu}, city={İstanbul}, country={Turkey}, year={2021}, pages={86-87} }