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Medetalibeyoglu A., Bahat G., Senkal N., Kose M., Avcı K., Sayın G., ...More
Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases, vol.89, pp.104717, 2021 (SCI-Expanded)
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Publication Type:
Article / Article
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Volume:
89
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Publication Date:
2021
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Doi Number:
10.1016/j.meegid.2021.104717
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Journal Name:
Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases
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Journal Indexes:
Science Citation Index Expanded (SCI-EXPANDED), Scopus, Academic Search Premier, BIOSIS, CAB Abstracts, EMBASE, Environment Index, Food Science & Technology Abstracts, MEDLINE, Veterinary Science Database
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Page Numbers:
pp.104717
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Keywords:
MBL2, Gene mutation, COVID-19, Hyperinflammation, Cytokine, SARS-CoV-2, C VIRUS-INFECTION, HEPATITIS-C, POLYMORPHISMS, CORONAVIRUS, SUSCEPTIBILITY, ASSOCIATION, SARS-COV-2, PROTEIN, MBL2
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Istanbul University Affiliated:
Yes
Abstract
Background/objectives: COVID-19 followed a mortal course in some young patients without any underlying factors, however, it followed a very benign course in some very older individuals with multiple comorbidities. These observations question if some genetic factors may be related to the vulnerability and poor prognosis of the disease. In this study, we aimed to investigate whether MBL2 gene B variant at codon 54 (rs1800450) were related to the variabilities in clinical course of this infection.